CASE 9524 Published on 20.09.2011

Torus palatinus


Head & neck imaging

Case Type

Clinical Cases


Eyselbergs M1,2, De Praeter G1,2, Dom M3, Vanhoenacker FM1,2,4

1. Department of Radiology, University Hospital Antwerp, Wilrijkstraat 10, B-2650 Edegem, Belgium
2. Department of Radiology, AZ Sint-Maarten, campus Duffel, Rooienberg 25, B-2570 Duffel, Belgium
3. Department of Oral and Maxillofacial Surgery, AZ Sint-Maarten, campus Duffel, Rooienberg 25, B-2570 Duffel, Belgium
4. Faculty of Medicine and Health Sciences, Ghent University, Belgium

71 years, female

Area of Interest Ear / Nose / Throat ; Imaging Technique CT
Clinical History
A 71-year-old woman with no past medical history was referred to the radiology department by the maxillofacial surgeon for preoperative evaluation of the maxilla for dental implant surgery. A multidetector computed tomography examination was performed and coronal (Fig. 1), sagittal (Fig. 2) and axial (Fig. 3) slices were reformatted.
Imaging Findings
A lobulated bony protuberance was seen at the midline of the hard palate. This osseous overgrowth consisted mainly of dense cortical bone and with only a small amount of cancellous bone in the centre of the lesion. The diagnosis of a torus palatinus was made.
A torus palatinus (TP) is a bony protuberance located at the union of the processus palatinus maxillaes that form the hard palate. This focal hyperostosis is covered by a thin and poorly vascularised layer of mucosa. Different phenotypes of the TP can be recognised with a flat, (poly)lobulated, spindle-shaped or nodular appearance. The lobulated or nodular presentation occurs mostly at both sides along the suture of the hard palate. The TP together with its mandibular counterpart, the torus mandibularis (TM), are the two most common forms of focal hyperostosis. A TM is typically located at the buccal side of the mandible above the mylohyoid line in the premolar region [1, 2]. The exact aetiology is still a matter of debate although a combination of genetic and environmental factors has been incriminated. Tori appear more frequently in certain ethnic groups such as Eskimos and the Japanese population. Also, a female predominance can be observed which renders the genetic factor more likely. The clinical onset of a TP is usually between the age of 11 and 20 years. A small TP is present in about 20% of the population and is mostly asymptomatic. A large TP can possibly lead to phonatory disturbances, limitation of mastication, food deposits and prosthetic instability [2]. Diagnosis is mostly incidental during a routine dental check-up since most tori are asymptomatic. When imaging is needed CT is the imaging modality of choice. Key points to diagnosis are the typical localisation of the lesion at the midline of the hard palate, the often symmetrical presentation along the midline and sharp delineation of the dense cortical bone with only a limited amount of cancellous bone in the centre.
When the palatal lesion is clinically suspect, the most important differential diagnosis that needs to be made is squamous cell carcinoma of the hard palate. In case of a TP the mucosal layer is intact [1].
A TP can also be seen as part of the clinical spectrum of the autosomal dominant inherited type of hyperostosis corticalis generalisata congenita also known as Worth syndrome [3]. This rare disease is characterised by increased bone density, palatal bony protuberances (TP) and thickening of various long bones. As tori are often an incidental finding during routine clinical examination, treatment is usually conservative and surgical removal is only performed in case of prosthetic instability or as a potential donor site for autogenous bone grafts [2].
Differential Diagnosis List
Torus palatinus
Squamous cell carcinoma of the hard palate
Sarcoma of the hard palate
Final Diagnosis
Torus palatinus
Case information
DOI: 10.1594/EURORAD/CASE.9524
ISSN: 1563-4086